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GHK-Cu (Copper Peptide)

Clinical10 studies

GHK-Cu is a naturally occurring copper-binding tripeptide found in human plasma, saliva, and urine, widely studied for its role in tissue remodeling, wound healing, and cellular regeneration.

Being studied for

  • Wound healing and skin regeneration
  • Collagen and elastin synthesis
  • Anti-inflammatory activity
  • Angiogenesis and tissue remodeling
  • Hair follicle and skin biology research

Published Research

Evidence profile

Predominantly in-vitro and preclinical; the controlled human trials identified reported no benefit over control

10 cited publications · 2 primary trials

Evidence gap: Two randomised controlled human trials of GHK-Cu as a discrete agent were identified — a 1992 venous ulcer trial and a 13-patient trial after laser resurfacing. Neither separated from its control on the objective endpoints. No controlled human trial in cosmetic skin ageing, and no systematic review or meta-analysis, was identified.

Findings below belong to the studies that reported them. They describe the material and conditions those authors used, not this product, and not an outcome to expect.

Core evidence

The publications most central to understanding this compound.

ClinicalRandomised controlled human trial with blinded evaluators

Effects of topical copper tripeptide complex on CO2 laser-resurfaced skin

Miller TR et al. · Archives of facial plastic surgery · 2006

Population: 13 patients who completed circumoral carbon dioxide laser skin resurfacing

Patients undergoing circumoral CO2 laser resurfacing were randomised to a post-treatment skin regimen with or without a copper tripeptide complex, with erythema quantified by computer software and by blinded evaluators, wrinkles and overall skin appearance assessed at 12 weeks, and patient-reported outcomes captured on a validated questionnaire.

Route reported
Topical skin care products applied to laser-resurfaced skin under the study protocol
Schedule
Post-procedure regimen applied as directed under the trial protocol
Duration
Assessment through 12 weeks after treatment under the study protocol
Treatment arms
Post-treatment skin care regimen containing GHK-Cu · Post-treatment skin care regimen without GHK-Cu
Blinded and software-based assessment of post-treatment erythema resolution
Computer analysis and blinded evaluators found no statistically significant differences between groups for earlier resolution of erythema.

Context: Only 13 patients completed the study, so it is very small and underpowered; the objective endpoints showed no between-group separation, the setting is post-procedure wound recovery rather than routine cosmetic use, and the only difference reported was on a subjective questionnaire.

PMID 16847171DOI 10.1001/archfaci.8.4.252View on PubMed
ClinicalProspective randomised, evaluator-blinded, placebo-controlled human trial

A prospective randomized evaluator-blinded trial of two potential wound healing agents for the treatment of venous stasis ulcers

Bishop JB et al. · Journal of vascular surgery · 1992

Population: 86 evaluable adult patients with chronic venous stasis ulcers

A randomised, evaluator-blinded trial compared two topical wound-healing agents against an inert vehicle placebo in adults with venous stasis ulcers, with change in ulcer size as the measured endpoint. The authors reported the outcome for a 0.4% tripeptide copper complex cream alongside a silver sulfadiazine comparator and the placebo arm.

Route reported
Topical application to the ulcer under the trial protocol
Duration
Trial duration not stated in the abstract
Treatment arms
Silver sulfadiazine 1% cream · Tripeptide copper complex 0.4% cream · Inert vehicle placebo cream
Comparison of ulcer size reduction between the tripeptide copper arm and placebo
There was no difference between the latter two treatments.

Context: The trial studied chronic venous leg ulcers, not cosmetic or ageing skin, and it reports a null comparison for the tripeptide copper arm relative to placebo; the abstract does not state the application schedule or treatment duration, and no cosmetic endpoints were measured.

PMID 1495150DOI 10.1067/mva.1992.37086View on PubMed
PreclinicalControlled in-vivo rat wound-chamber study with companion rat dermal fibroblast cultures

Expression of glycosaminoglycans and small proteoglycans in wounds: modulation by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu(2+)

Siméon A et al. · The Journal of investigative dermatology · 2000

Population: Sprague-Dawley rats with implanted subcutaneous wound chambers, plus cultured rat dermal fibroblasts

Using a rat subcutaneous wound-chamber model with parallel rat dermal fibroblast cultures, the authors measured wound tissue dry weight and total protein, type I collagen and glycosaminoglycan content, electrophoretic glycosaminoglycan profiles, and decorin and biglycan mRNA over a 22-day time course in chambers injected with the tripeptide-copper complex versus controls.

Route reported
Direct injection into the implanted subcutaneous wound chamber under the study protocol
Schedule
Repeated injections under the study protocol
Duration
Chambers analysed at intervals through day 22 under the study protocol
Treatment arms
Wound chambers receiving repeated GHK-Cu injections of 2 mg per injection · Control wound chambers
Change in decorin and biglycan mRNA levels in treated wound chambers
Glycyl-histidyl-lysine-Cu(2+) treatment increased the mRNA level of decorin and decreased those of biglycan.

Context: A rodent implanted-chamber model with direct injection of a milligram-scale dose, which is not comparable to topical cosmetic application in humans; it measures biochemical and mRNA markers of matrix turnover, not any clinical or appearance endpoint.

PMID 11121126DOI 10.1046/j.1523-1747.2000.00166.xView on PubMed
PreclinicalIn-vitro concentration-response study in cultured fibroblasts

Stimulation of collagen synthesis in fibroblast cultures by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu2+

Maquart FX et al. · FEBS letters · 1988

Population: Cultured fibroblasts; the abstract does not name the donor species and the record is indexed under Humans

This early laboratory study measured collagen synthesis in fibroblast cultures exposed to the tripeptide-copper complex across a range of concentrations, alongside cell number, and noted the presence of a GHK triplet in the alpha-2(I) chain of type I collagen as a proposed origin of the peptide.

Route reported
Added to the fibroblast culture medium in vitro
Treatment arms
Fibroblast cultures exposed to GHK-Cu across a concentration range of approximately 10(-12) to 10(-9) M · Untreated culture comparison
Concentration range over which collagen synthesis changed in culture
The stimulation began between 10(-12) and 10(-11) M, maximized at 10(-9) M, and was independent of any change in cell number.

Context: A cell-culture experiment only: no animal or human skin was studied, the concentrations tested are laboratory conditions rather than anything achieved by topical application, and the abstract does not state the fibroblast donor species.

PMID 3169264DOI 10.1016/0014-5793(88)80509-xView on PubMed

Supporting evidence

PreclinicalEx-vivo human skin permeation study using flow-through diffusion cells

Human skin penetration of a copper tripeptide in vitro as a function of skin layer

Hostynek JJ et al. · Inflammation research : official journal of the European Histamine Research Society ... [et al.] · 2011

Population: Excised human skin preparations: isolated stratum corneum, heat-separated epidermis and dermatomed skin

This laboratory study applied an aqueous copper tripeptide solution to three excised human skin preparations in flow-through diffusion cells and used inductively coupled plasma mass spectrometry to quantify copper in the receptor fluid and retained in tissue over 48 hours, deriving a permeability coefficient for dermatomed skin.

Route reported
Topical application to excised human skin in flow-through diffusion cells (1 cm2 exposure area)
Schedule
Single infinite-dose application under the study protocol
Duration
48 hours, with receptor fluid collected at 4-hour intervals
Treatment arms
0.68% aqueous copper tripeptide applied under infinite dose conditions
Permeability coefficient and 48-hour copper flux through dermatomed human skin
The permeability coefficient of the compound through dermatomed skin was 2.43 ± 0.51 × 10(-4) cm/h; 136.2 ± 17.5 μg/cm(2) copper permeated 1 cm(2) of that tissue over 48 h, while 97 ± 6.6 μg/cm(2) were retained as depot.

Context: Excised skin in diffusion cells under infinite-dose conditions is not living skin and has no circulation or metabolism; the assay tracks copper rather than the intact tripeptide, and the authors' framing is transdermal anti-inflammatory delivery, not cosmetic use.

PMID 20721598DOI 10.1007/s00011-010-0238-9View on PubMed

Safety literature

Reports of adverse events and individual cases. A case report describes what happened to one person; it cannot establish cause, and it cannot establish how often anything occurs.

PreclinicalIn-vitro comparative skin irritation screening in a keratinocyte model

Selected Biomarkers Revealed Potential Skin Toxicity Caused by Certain Copper Compounds

Li H et al. · Scientific reports · 2016

Population: Cultured human keratinocytes (cell-line based skin irritation model)

This comparative laboratory study treated cultured human keratinocytes with three copper compounds and measured cell viability by tetrazolium reduction, cytotoxicity by lactate dehydrogenase release, and expression of irritation-related biomarkers (including IL-1 alpha, IL-8, HSPA1A and FOSL1) by real-time PCR and protein quantification.

Route reported
Compounds added to keratinocyte culture medium in vitro
Schedule
24-hour treatment under the study protocol
Duration
24 hours, at test concentrations including 58 and 580 micromolar
Treatment arms
Copper peptide (GHK-Cu) · Copper chloride (CuCl2) · Copper acetate (Cu(OAc)2)
Cytotoxicity and irritation-biomarker response to GHK-Cu relative to inorganic copper salts
GHK-Cu was not cytotoxic and did not induce any significant change in the expression levels of various skin irritation-related biomarkers.

Context: A monolayer keratinocyte screening assay, not a human patch test or clinical safety study; it addresses local irritation signals only and says nothing about systemic safety, sensitisation, or repeated real-world use.

PMID 27892491DOI 10.1038/srep37664View on PubMed
Additional indexed literature (4)

Published studies summarised here concern the specific investigational or pharmaceutical materials and study conditions described by their authors. Their findings are not performance claims for Cobalt Peptides material. Citations verified against PubMed on 2026-08-09.

GHK-Cu has been extensively studied in laboratory and cosmetic research contexts. It is not approved as a medicinal product for therapeutic use.

Notice

All information on this page is provided strictly for laboratory research and educational purposes only.